@article {685,
	title = {Management of Choroidal Granulomas Involving the Macula in Corticosteroid-Intolerant Patients.},
	journal = {JAMA Ophthalmol},
	volume = {133},
	year = {2015},
	month = {2015 Nov},
	pages = {1351-2},
	keywords = {Adult, Choroid Diseases, Combined Modality Therapy, Dexamethasone, Drug Therapy, Combination, Drug Tolerance, Folic Acid Antagonists, Glucocorticoids, Granuloma, Humans, Interferon-gamma Release Tests, Intravitreal Injections, Macula Lutea, Male, Methotrexate, Tuberculoma, Tuberculosis, Ocular, Vitrectomy},
	issn = {2168-6173},
	doi = {10.1001/jamaophthalmol.2015.1951},
	author = {Chin, Eric K and Almeida, David R P and Mahajan, Vinit B}
}
@article {115,
	title = {Calpain-5 mutations cause autoimmune uveitis, retinal neovascularization, and photoreceptor degeneration.},
	journal = {PLoS Genet},
	volume = {8},
	year = {2012},
	month = {2012},
	pages = {e1003001},
	abstract = {<p>Autosomal dominant neovascular inflammatory vitreoretinopathy (ADNIV) is an autoimmune condition of the eye that sequentially mimics uveitis, retinitis pigmentosa, and proliferative diabetic retinopathy as it progresses to complete blindness. We identified two different missense mutations in the CAPN5 gene in three ADNIV kindreds. CAPN5 encodes calpain-5, a calcium-activated cysteine protease that is expressed in retinal photoreceptor cells. Both mutations cause mislocalization from the cell membrane to the cytosol, and structural modeling reveals that both mutations lie within a calcium-sensitive domain near the active site. CAPN5 is only the second member of the large calpain gene family to cause a human Mendelian disorder, and this is the first report of a specific molecular cause for autoimmune eye disease. Further investigation of these mutations is likely to provide insight into the pathophysiologic mechanisms of common diseases ranging from autoimmune disorders to diabetic retinopathy.</p>
},
	keywords = {Amino Acid Sequence, Base Sequence, Calpain, Cell Line, Cells, Cultured, Choroid Diseases, Exome, Exons, Eye Diseases, Hereditary, Female, Gene Expression, Genetic Linkage, Humans, Male, Models, Molecular, Molecular Sequence Data, Mutation, Pedigree, Phenotype, Photoreceptor Cells, Vertebrate, Protein Conformation, Protein Transport, Retinal Degeneration, Sequence Alignment},
	author = {Mahajan, Vinit B and Skeie, Jessica M and Bassuk, Alexander G and Fingert, John H and Braun, Terry A and Daggett, Heather T and Folk, James C and Sheffield, Val C and Stone, Edwin M}
}
@article {777,
	title = {Re: Intraoperative choroidal detachment during 23-gauge vitrectomy.},
	journal = {Retina},
	volume = {32},
	year = {2012},
	month = {2012 Feb},
	pages = {411},
	keywords = {Choroid Diseases, Drainage, Humans, Incidence, Intraoperative Complications, Microsurgery, Retinal Detachment, Retinal Perforations, Risk Factors, Vitrectomy},
	issn = {1539-2864},
	doi = {10.1097/IAE.0b013e3182398122},
	author = {Tarantola, Ryan M and Mahajan, Vinit B}
}
